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Welcome

I created this blog as an instrument of what I have encountered in the world of veterinary medicine as a proud vet student. Comments and suggestions are welcome here at;

sweet_daffodil90@yahoo.co.uk

Regards,
Aina Meducci 2012

Disclaimer

The following blog posts is not genuinely from my research but through readings and citation from trusted website. I do not own any of the copyright and therefore you may use it at your own risk

SINCE I AM NOT A VETERINARIAN YET, THEREFORE I CAN'T CONSULT ANY MEDICAL ADVICE TO YOU AND YOUR PETS! EXTREMELY IMPORTANT!.

Happy reading!
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Malocclusion in Horse

A friend of mine poked me on facebook asking about malocclusion in horses. Frankly speaking this is my first time to read the word. I did not know anything about equine dentistry, but maybe I can learn something while googling this info. Thanks Azamin :)

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Malocclusion in horse

An equine malocclusion is a misalignment between the upper and lower jaws of a horse or other equine. It results in a faulty bite with the upper and lower teeth failing to meet correctly. Malocclusions can cause pain to the horse and may also lead to weight loss and other eating problems related to poor chewing or loss of appetite especially in ageing horse.

It is also known as the inability of the arcades of molars or incisors to occlude, fit together properly or work effectively/efficiently.


Parrot jaw


Normal presentation of teeth of 10 years old horse


Below are the common equine dental malocclussion

MOLARS

1. Caudal hooks


Definition - Dominant lower or upper last molar overhanging opposing molar.
Etiology - Hereditary - May result from horse born with over or under bite.
Developmental - May result secondary to molar malocclusion that forces jaw out of alignment.

Resulting Problems - prevents horse from chewing freely side to side resulting improper and excessive molar wear. Tight soft tissues are very susceptible to lacerations from sharp molar points. Can cause extreme discomfort as head carriage or frame is changed when riding.

Solution - Reduce dominant molar to prevent excessive wear to opposing molar and carefully round to prevent soft tissue trauma.


2. Rostral hooks



Definition - Dominant upper front premolars overhang lower premolars.
Etiology - Hereditary - May result from horse born with an overbite.
Developmental - May result secondary to molar malocclusion that forces jaw out of alignment.

Resulting Problems - Prevents horse from chewing freely side to side resulting in improper and excessive molar wear. Can cause excessive wear to opposing lower premolar. Can cause severe bit problems and trauma to soft tissue.

Solution - Reduce dominant portion of upper premolar, rounding unopposed portion of premolar smoothly toward gum. Requires preventative maintenance to prevent reoccurrence.


3. Ramps


Definition - Excessive height to lower premolars.
Etiology
- May occur when upper from premolar deciduous cap, or baby tooth is retained preventing normal growth of permanent premolar. Can occur if a tooth is reduced without corresponding correction to opposing tooth.

Resulting Problem - Prevents horse from chewing freely side to side resulting in improper and excessive molar wear. Can cause severe discomfort with the bit. Can force lower jaw forward over time causing under bite.

Solution - Reduce dominant tooth to allow opposing too the recover over time.


4. Enamel points

Definition - sharp points that generally form on the outside of the upper molars and the inside of the lower molars.
Etiology - Occurs over time as horse stops chewing as far sideways resulting in unopposed edge tooth getting longer and the enamel forms razor sharp points.

Resulting Problem
- Enamel points traumatize soft tissues causing pain and discomfort when eating and also from bits and other head gear pulling cheeks and tongue into these areas. Also stops horse from chewing freely sideways.

Solution - Remove sharp enamel points.


5. Sheared molar table


Definition - Extreme angulation of chewing surface of molars.
Etiology - Results from lack of lateral excursion or side to side chewing.

Resulting Problems - Very severe problem. Does not allow horse to chew sideways resulting in very poor utilization of feed.

Solution - Correct molar table angle of upper and lower molars over time allow horse freedom to chew side to side.


6. Wave complexes



Definition - Molar arcade develops an uneven "wavelike" appearance generally involving many premolars and molars.

Etiology - Often occurs secondary to other malocclusions such as retained deciduous caps, missing teeth, hooks, ramps, etc. Results from horse being unable to obtain free side to side chewing.

Resulting Problems - Causes gradual excessive wear to many molars, resulting in prematurely worn out teeth, periodontal pocketing, decay and loss of teeth. Prevents free side to side chewing and inhibits proper grinding of feed.

Solution - Reduce high complexes to allow recovery of opposing teeth and allow freedom to chew freely side to side to encourage proper use. Requires preventative maintenance over time.


7. Stepped molars


Definition - Molar arcade develops stepped-like appearance.
Etiology - Most often occurs due to a missing tooth allowing the opposing tooth to grow into the void. Can occur from extreme uneven wear. May occur when a deciduous cap or baby tooth is retained too long delaying normal growth of permanent tooth and allowing opposing tooth to grow too long.

Resulting Problems - Prevents or blocks the horse from being able to chew freely resulting in uneven and improper wear to molars and incisors.

Solution - Reduce high molar to allow freedom to chew correctly. Requires maintenance to prevent unopposed tooth from growing into space.


8. Accentuated transverse ridges


Definition - Enlarged ridges that run across chewing surfaces of molars.
Etiology - Can result from a shift in jaw alignment. Forms from a harder area in one tooth over powering and wearing into a soft area in an opposing tooth. also can occur if horse is not chewing in proper sideways motion.

Resulting Problems - Interfere with normal chewing and jaw motion front to back. Cause excessive wear to opposing molars. Can result in severe periodontal disease and diastema.

Solution - Reduce enlarged ridges to allow opposing teeth to recover and horse to chew correctly. Will improve when existing malocclusions are corrected.


9. Periodontal pockets



Definition - Gum disease around tooth causing an area for feed to pocket.
Etiology - Results from malocclusion or misalignment of teeth. Progresses as horse is unable to chew correctly.

Resulting Problem - Gum disease leads to bone erosion around teeth and eventually can cause infection, abscess and loss of tooth.

Solution - Correct malocclusion as early as possible. Very difficult to stop progression of periodontal disease if it is not stopped early on. Prevention is the key.


9. Diastema



Definition - Space that occurs between two adjoining teeth.
Etiology - Often results as malocclusions force two teeth apart over time.

Resulting Problems - Causes feed to get trapped between teeth resulting in progressive and severe periodontal disease. Results in overall poor oral health.

Solution - Very difficult to correct. Reduce unopposed overgrowth of opposing teeth. Prevention of malocclusions in th key.


INCISORS

1. Overbite



Definition - Upper incisors protrude in front of lower incisors.

Etiology - Hereditary - Horse may be born with an overbite. This will often result in rostral and/or caudal hooks. Developmental - Rostral and/or caudal hooks can shift alignment of bite resulting in an overbite.

Resulting Problem - Prevents horse from chewing freely side to side resulting in improper and excessive molar wear. Rostral and caudal hooks, transverse ridges, wave complexes and sheared molar table angles will become more severe as the horse is forced to chew incorrectly over time. Can also cause severe discomfort with the bit.

Solution - Reduce length of upper incisors. Correct molar malocclusions.


2. Underbite


Definition - Lower incisors protrude in the front of upper incisors.
Etiology - Hereditary - Horse is born with under bite. Developmental - Ramps on lower premolars can cause lower jaw to shift forward resulting in an under bite.

Resulting Problems - Prevents horse from chewing freely side to side resulting in improper and excessive molar wear. Ramps, transverse ridges, wave complexes and sheared molar table angles will become more severe as horse is forced to chew incorrectly over time. Can also cause severe discomfort with the bit. Can result in TMJ pain.

Solution - Reduce length of lower incisors. Correct corresponding molar abnormalities.


3. Ventral curvature (smile)


Definition - Outer corner lower incisors grow longer relative to outer corner upper incisors.
Etiology - May occur when deciduous caps are rretained onupper corner incisors preventing normal growth of permanent incisors. Can occur due to abnormal chewing secondary to molar malocclusions.

Resulting Problems - Inhibits horse from chewing freely side to side often resulting in improper and excessive molar wear.

Solution - Reduce length of upper central incisors and lower corner incisors to allow free side to side movement.


4. Dorsal curvature (frown)



Definition - Outer corner upper incisors grow longer relative to outer corner lower incisors.
Etiology - May occur when deciduous caps are retained on lower corner incisors preventing normal growth of permanent incisors. Often occurs from cribbing and wwoodchewing and can occur due to abnormal chewing secondary to molar malocclusions.

Resulting Problems - Inhibits horse from chewing freely side to side often resulting in improper and excessive molar wear.

Solution - Reduce length of upper corner and lower central incisors to allow free side to side movement.


5. Offset or diagonal bite


Definition - Upper incisors on one side of mouth are excessively long and lower incisors on opposite side are excessively long, causing incisors to meet on a diagonal.

Etiology - May develop over time if horse chews more in one direction. Can result from cribbing and wood chewing and may occur secondary to molar malocclusion or as a result of a missing incisor or incisor damage causing opposing incisors to become excessively long.

Resulting Problem - Causes horse to excessively wear molars on one side of mouth and opposite side molars become excessively long resulting in a sheared molar table angle. Can result in TMJ pain.

Solution - Reduce excessively long incisors. Correct corresponding molar mmalocclusions Will need frequent follow up corrections.


6. Bone spur


Definition - Mandibular periotitis or abnormal bony growth due to trauma.
Etiology - Most often caused by trauma from bit to lower jaw bone.

Resulting Problem - Can be extremely painful causing behavioral problems when bit contacts area.

Solution - Surgical removal of affected bone.


7. Wolf tooth



Definition - Small shallow rooted teeth in front of premolars. Most common in upper jaw. Can occasionally occur in front of lower premolars.

Etiology - A normal finding in most horses. May not appear in some horses.

Resulting Problem - Bit may contact tooth causing severe pain and discomfort as tooth moves. Can fracture or become loose causing discomfort

Solution - Wolf teeth should be removed before horse starts being bitted to prevent discomfort and bad habits.


8. Blind wolf tooth


Definition - Abnormal wolf tooth that does not break through gums.

Etiology - Blind Wolf Tooth usually comes in at an angle, often up to one inch forward of premolars. Angle of tooth does not allow it to penetrate gums.

Resulting Problem - Can cause severe bit problems. In a location that bit often contacts. Often missed due to being buried under gums.

Solution - Generally easy to surgically remove.


Source: Equine health from discerning hands equine dentistry; common equine dental malocclusions

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Ruminants and rumination

I found this interesting information while reading a physiology book about ruminant. Up until this moment, I was wondering the process of rumination instead of regurgitation, remastication and all. Without knowing the true facts, you'd only know the basic concept of rumination, not the whole process. I was surprised to know that I have not encountered this chapter so far, even though we have left ruminant class for a year. So, I think it's not too late to go back while we still have a time recap rumination, simple definition but hard to tell. ;)

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Ruminants (who are they?)

A ruminant is a mammal of the order Artiodactyla that digests plant-based food by initially softening it within the animal's first stomach, then regurgitating the semi-digested mass, now known as cud, and chewing it again.

There are two suborder of ruminants;

1) Ruminantia

Deer, moose, elk, sheep, cow, goat, reindeer, caribou, antelope, giraffe, musk ox, bison

Caribou antelope

musk ox bison


2) Tylopoda: camel, llama, alpaca, vicuna


alpaca llama

vicuna camel


DIFFERENCE BETWEEN RUMINANTIA AND TYLOPODA??


1) Tylopoda do not have OMASUM


Camel digestive system

2. Tylopoda have areas of cardiac glands that open into ventral sacculated surfaces of the reticulum and rumen


The ruminant stomach


Composed of 4 compartments

  1. Rumen (paunch)
  2. Reticulum (honeycomb)
  3. Omasum (many plies)
  4. Abomasum (true stomach)

Rumen Reticulum


Omasum Abomasum


The ruminant stomach adapted for fermentation of ingested food by bacterial and protozoan microorganism. Energy is obtained through fermentation. Diet of ruminants includes mostly of growing, mature and dried grass and mammalian enzymes cannot digest the cellulose in these materials. However, microbial enzymes can digest the plant cell through fermentation process

The first 3 compartments are also known as forestomach because they precede the true stomach (abomasum). The rumen occupies a prominent portion of the viscera on the left side while reticulum is proximity to the heart as seen on the right side. The abomasum is mostly on the right side.


The position of the stomach's compartments of ruminant


Te abomasum is the largest compartment of the newborn ruminants while in adult ruminant, rumen is the largest forestomach compartment.

When the food (bolus) is passed down from the esophagus, it is deposited in the cranial sac of rumen, the next contraction transfers the contents to the reticulum whereby they can be pumped by reticulum contraction;

  1. to cardiac opening for regurgitation
  2. to the omasum through reticulomasum
  3. to caudal parts of rumen

Densed objects are always retained in the reticulum and may cause hardware disease (traumatic pericarditis)- penetrate to heart or lungs


Summary of ruminants stomach

Rumen

Allows soaking and fermentation of bulk fibrous food and mixing of food

Reticulum

Serves as pump that cause liquid to flow into and out of rumen. The flow of liquid directs ingesta into the rumen, regulates its passage from rumen to omasum, supplies moisture to rumen and floods to cardia(esophagus) before regurgitation

Omasum

Continued fermentation and absorption and regulation of onwards propulsion between reticulum and abomasum.

Abomasum

True stomach function. Digestion of degraded roughages and concentrates begins for fermentation residues that have not been already absorbed.



Rumination Process

The process of bringing the food material back from the ruminant stomach to the mouth for further remastication (chewing). This is the ruminant's strategy to increase the surface area of the chewing feed for maximum absorbtion to be converted as volatile fatty acid (acetic acid, propionate, and butyric acid) for energy and metabolism of ruminant's body

Composed of 4 phases

  1. Regurgitation
  2. Remastication
  3. Resalivation
  4. Redeglutition (reswallow)

Rumination cycle starts with regurgitation of bolus. Regurgitation is done by taking a breath with closed glottis (opening to trachea) which causes

  1. Thoracic cavity enlarges without lung inflation
  2. Intrapleural pressure decrease and lowering pressure in the mediastinal space and organs located within it (eg esophagus)
  3. Cardia (caudal of esophagus) opens due to lower pressure of esophagus and rumen contents is brought out to the esophagus.
  4. Reverse peristalsis is initiated and food mass bolus is quickly carried to the mouth.
  5. Reticulum contracts just before regurgitation to ensure a rumen mixture in the region of cardia. It also clears the cardia of recently swallowed boluses.
(the regurgitation process may be seen on the left sided of neck)

After the regurgitated bolus arrives in mouth, the liquid is squeezed from it and swallowed. Remastication and resalivation occurs simultaneously. The number of chew given to each bolus depending on diet. Redeglutition occurs appropriate time and the next cycle of ruminantion iccurs in 5 second. The

The time spent in rumination each day is varies with species and diet. The more coarse the feed, the more time spend for rumination.

The gases produced in the rumen as a result or fermentation are mainly carbon dioxide (60-70%) and methane (30-40%). Nitrogen, oxygen, and hydrogen present in trace amount as intermediate for other reactions. Carbon dioxide is produce during the fermentation of carbohydrates. and deamination of amino acid and also produce from salivary bicarbonate when it neutralizes the fatty acids produced from microbial fermentation of lipids. Methane is formed by the reduction of carbon dioxide by methane-producing bacteria.

Eructation is the process by which gas from the forestomach is removed by way of the esophagus to the pharynx. It is occurs every 1 minute. The primary stimulus for eructation is the presence of gas in the dorsal sac. If the gas artificially placed into the dorsal rumen, the frequency and volume of eructation increases.


Source; Functional anatomy and physiology of domestic animals, 4th ed, William O Reece, Wiley-Blackwell 2009

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Chick embryo development

It's all started within an egg



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Infertile egg
(no development)



Day 1- day 2
Appearance of tissue development



Day 2- day 3
Heart beat and blood vessels clearly visible



Day 4
Eye pigmented

Day 5
Appearance of elbows and knees

Day 6-day 7
Appearance of beak and voluntary movement begins
Comb growth begins and egg tooth begins to appear

Day 8
Feather tract seen and upper and lower beak equal in length

Day 9
Embro starts to look bird-like and mouth opening appears

Day 10
Egg tooth prominent and development of toe nails

Day 11
Comb serrated and tail faethers apperent

Day 12
Toes fully formed and first few visible feathers

Day 13
Appearance of scale and body covered slightly with feathers

Day 14
Embryo turns head towards large end of egg

Day 15
Gut is drawn into abdominal cavity

Day 16
Feathers cover complete body and albumen completely gone

Day 17
Amniotic fluid decreases and head is between legs

Day 18
Growth of embryo nearly complete, yolk sac still outside embryo, and head is under right wing

Day 19
Yolk sac draws into abdominal cavity, amniotic fluid gone and embryo occupies most of space within egg (not in the air cell)

Day 20
Yolk sac drawn completely into body, embryo becomes a chick (breathing in air cell), and internal and external pip

Day 21
A complete chick (hatched and dried)


Sources: Cobb hatchery embryo development


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Important Poultry Disease (Bacterial and fungal diseases)

Recently coming back after finishing my practical training in broiler breeder farm, and it's time to get serious!

Today's post is the continuation from the previous post about the common poultry diseases found in chickens. Note that there are many other thousand of poultry diseases but this post is just a remark of what I have learnt during my practical training. You may refer to other website for more details regarding diseases.

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1. Chronic Respiratory Disease

  • Mycoplasma gallisepticum
  • Respiratory distress and inflammation of the airsac often occur
Caseous exudate is frequently observed on the airsacs of birds infected with Mycoplasma gallisepticum

  • pericarditis, peritonitis and perihepatitis

2. Infectious synovitis

  • Mycoplasma synoviae
  • Often affects joints and hock




3. Fowl cholera (avian paseurellosis)

  • Swelling of wattles and comb



4. Parathyroid infection

  • Cause by salmonella bacteria (S.typhimurium)
Button type lesion in the chicken's intestine affected with paratyphoid infection


5. Pullorum disease

  • Salmonella pollurum
  • Ovaries are dark in color
  • Diarrhea and septicaemia occur followed by quick death

Fowl typhoid. Enlarged, bronzed to the mahogany colour liver and enlarged, mottled and brittle spleen in a turkey Diseased liver and spleen are shown in contrast to the normal ones at left.


6. Infectious coryza

  • Haemophilus paragallinarum
  • cause facial edema
Facial edema

7. Aspergillus (fungal disease)

  • Respiratory infection caused by fungus inhaled from mouldy litter
  • Affects mostly to young chickens
Lung nodules in chickens affected with aspergillosis


source: FFM Farms Sua Betong PD

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